Toxicological Profile for Cyanide
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Toxicological Investigation of Acute Cyanide Poisoning of a 29-year-old Man: A Case Report
Background: Cyanide poisoning is frequently lethal, Because of the early onset of severe symptoms and difficulty in the diagnosis. Case: The case was a 29-year-old boy who collapsed suddenly after taking a bottle of beer in a bar. Samples, such as peripheral blood, stomach contents, bile fluid, urine and mouth swabs, were prepared using standard autopsy procedure and were subjected to analysis...
متن کاملHydrogen Cyanide and Cyanide Salts (CASRN Various) | IRIS | US EPA
Note: A TOXICOLOGICAL REVIEW is available for this chemical in Adobe PDF Format (153 pp, 905K). Similar documents can be found in the List of Available IRIS Toxicological Reviews. Links to specific pages in the toxicological review are available throughout this summary. To utilize this feature, your Web browser and Adobe program must be configured properly so the PDF displays within the browser...
متن کاملInteraction between noise and asphyxiants: a concern for toxicology and occupational health.
The article highlighted in this issue is "Potentiation of Noise-Induced Hearing Loss by Low Concentrations of Hydrogen Cyanide in Rats" by Laurence D. Fechter, Guang-Di Chen, and David L. Johnson (pp. 131-138).
متن کاملInteraction of cyanide and nitric oxide with cytochrome c oxidase: implications for acute cyanide toxicity.
Acute cyanide toxicity is attributed to inhibition of cytochrome c oxidase (CcOX), the oxygen-reducing component of mitochondrial electron transport; however, the mitochondrial action of cyanide is complex and not completely understood. State-3 oxygen consumption and CcOX activity were studied in rat N27 mesencephalic cells to examine the functional interaction of cyanide and nitric oxide (NO)....
متن کاملp38 Mitogen-activated protein kinase regulates Bax translocation in cyanide-induced apoptosis.
Execution of cyanide-induced apoptosis is mediated by release of cytochrome c from mitochondria. To determine how cyanide initiates cytochrome c release, Bax translocation was investigated in primary cultures of cortical neurons. Under nonapoptotic (control) conditions, Bax resided predominantly in the cytoplasm. After 300-microM cyanide treatment for 1 h, Bax translocated to the mitochondria, ...
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تاریخ انتشار 2006